Clinical research & concentration reference

Survodutide Research & Reconstitution: BAC Water, Studies & Evidence

Survodutide (BI 456906) is an investigational dual glucagon receptor and GLP-1 receptor agonist being studied in obesity, type 2 diabetes, metabolic dysfunction-associated steatohepatitis (MASH), and related metabolic conditions. Phase 2 and emerging phase 3 evidence provide substantial human clinical data, but survodutide is not presented here as an FDA-approved medicine or as having an FDA-approved human dosage.

INVESTIGATIONAL CLINICAL EVIDENCE RESEARCH CONCENTRATION PHASE 3 DATA
Regulatory distinction: Survodutide is an investigational compound. The clinical-trial doses described on this page are study regimens, not FDA-approved dosages, prescribing recommendations, or personalized treatment instructions.

Clinical Research Snapshot

Compound Survodutide (BI 456906)
Drug / compound class Dual glucagon receptor (GCGR) and GLP-1 receptor agonist
Mechanism Concurrent agonism of glucagon and GLP-1 receptors
U.S. regulatory status Investigational; no FDA-approved indication established
Clinical development phase Phase 3 clinical development, with phase 2 efficacy and safety evidence published
FDA-approved indication None established for survodutide
Human evidence level Moderate-to-high and expanding, including randomized phase 2 trials and published phase 3 obesity results
Administration route studied Primarily subcutaneous administration in clinical trials
Major research areas Obesity, type 2 diabetes, MASH, liver fibrosis and metabolic disease
Last scientifically reviewed September 11, 2026

Survodutide Mechanism & Clinical Research Overview

Survodutide Dual-Receptor Activity GCGR glucagon receptor GLP-1R GLP-1 receptor Survodutide Dual glucagon + GLP-1 receptor agonism
Simplified educational illustration of survodutide's two primary receptor targets.
Survodutide Research Program Survodutide BI 456906 Obesity Phase 2 + Phase 3 Type 2 Diabetes Phase 2 + Phase 3 MASH Phase 2 evidence Liver / Fibrosis Ongoing investigation
Major human research areas represented in the survodutide development program.

Survodutide BAC Water & Concentration Reference

Survodutide is investigational and does not have an FDA-approved commercial labeling framework establishing a universal BAC-water reconstitution procedure for a research vial.

Clinical-trial publications describe study formulations and administration procedures, but those study-specific formulations should not automatically be treated as instructions for an unrelated research material.

Important formulation distinction. BacScience does not present BAC-water mixing as an FDA-approved preparation method for survodutide. The concentration examples below are mathematical references only and are not human dosing, administration, or treatment protocols.

Research Concentration Examples — Mathematical Reference

The governing equation is:

Total vial quantity ÷ final liquid volume = concentration

Example: a hypothetical research vial containing 10 mg of compound with a final liquid volume of 2 mL corresponds mathematically to a concentration of 5 mg/mL.

Research vial quantity Final liquid volume Calculated concentration Mathematical reference
5 mg 1 mL 5 mg/mL Quantity divided by volume
5 mg 2 mL 2.5 mg/mL Quantity divided by volume
10 mg 1 mL 10 mg/mL Quantity divided by volume
10 mg 2 mL 5 mg/mL Quantity divided by volume
15 mg 1.5 mL 10 mg/mL Quantity divided by volume
30 mg 3 mL 10 mg/mL Quantity divided by volume
Mathematical reference only: A calculated concentration does not establish sterility, stability, compatibility, biological activity, dosing suitability, or an appropriate preparation method.

Calculate a Custom Concentration

Use the BacScience universal BAC-water calculator for concentration mathematics.

Open BAC Water Calculator

What Is Survodutide?

Survodutide, also known as BI 456906, is an investigational peptide designed to activate both the glucagon receptor and the glucagon-like peptide-1 receptor.

Unlike selective GLP-1 receptor agonists, survodutide combines GLP-1-receptor activity with glucagon-receptor activity. This dual mechanism is being investigated for effects on body weight, glycemic control, energy metabolism and liver-related endpoints.

Human clinical development has included randomized phase 2 studies in obesity, type 2 diabetes and MASH, followed by phase 3 programs in obesity and obesity with type 2 diabetes.

Mechanism of Action

Survodutide is a dual agonist of the glucagon receptor (GCGR) and GLP-1 receptor (GLP-1R). The GLP-1 component is associated with incretin-related metabolic effects, while glucagon-receptor activity is being investigated for effects on energy expenditure, lipid metabolism and other metabolic pathways.

The combined receptor activity is one reason survodutide has been investigated beyond glucose control, including in obesity and metabolic dysfunction-associated steatohepatitis.

Scientific distinction: Mechanistic rationale does not establish clinical efficacy or regulatory approval. Those questions require controlled human clinical evidence.

FDA / Regulatory Status

Survodutide is investigational. It is not presented on this page as an FDA-approved medication and there is no FDA-approved survodutide dosage or labeled indication to report.

Investigational does not mean unstudied. Survodutide has progressed through randomized phase 2 studies and into phase 3 development. However, clinical-trial evidence and an investigational dose remain distinct from FDA approval.

Approved Dosage vs. Doses Studied in Clinical Trials

FDA-approved dosage None. Survodutide does not currently have an FDA-approved dosage or FDA-approved indication.
Doses studied in trials Clinical studies have evaluated different dose ranges and escalation schedules depending on indication and protocol, including once-weekly regimens in obesity, diabetes and MASH studies.
Important: A dose appearing in a phase 2 or phase 3 study is a study parameter. It is not automatically a recommended dose for an individual.

Survodutide Clinical Development History

Early clinical studies evaluated survodutide in metabolic disease, including type 2 diabetes. Subsequent phase 2 programs examined obesity and MASH, generating evidence for both metabolic and liver-related outcomes.

The obesity program progressed into phase 3 SYNCHRONIZE studies. Published 2026 results from SYNCHRONIZE-1 reported significant weight reduction with 3.6 mg and 6.0 mg survodutide compared with placebo over 76 weeks.

Phase 3 development also includes SYNCHRONIZE-2 in participants with overweight or obesity and type 2 diabetes.

Major Human Clinical Trials

The following table emphasizes randomized human studies and current clinical-development evidence. Trial doses are reported for research interpretation only.

Trial Phase Population N Dose studied Route Frequency Duration Primary outcome / key finding Source
Phase 2 T2D
NCT04153929
2 Type 2 diabetes on metformin 413 randomized Up to 0.3, 0.9, 1.8 or 2.7 mg once weekly; additional twice-weekly groups SC Weekly / twice weekly 16 weeks Survodutide reduced HbA1c and body weight, with dose-related effects and predominantly gastrointestinal adverse events. PubMed
Obesity Phase 2
NCT04667377
2 Overweight / obesity without diabetes 387 enrolled 0.6, 2.4, 3.6, 4.8 mg SC Once weekly 46 weeks Mean body-weight changes were −6.2%, −12.5%, −13.2% and −14.9% for the 0.6, 2.4, 3.6 and 4.8 mg groups, respectively, versus −2.8% with placebo. PubMed
MASH Phase 2
NCT04771273
2 Biopsy-confirmed MASH with F1–F3 fibrosis 293 2.4, 4.8, 6.0 mg SC Once weekly 48 weeks Histologic improvement in MASH without worsening of fibrosis occurred more frequently in survodutide groups than placebo. PubMed
SYNCHRONIZE-1
NCT06066515
3 Obesity / overweight without type 2 diabetes 726 3.6 and 6.0 mg SC Once weekly 76 weeks Mean body-weight change was −12.2% with 3.6 mg and −13.0% with 6.0 mg versus −5.4% with placebo at week 76. NEJM / PubMed
SYNCHRONIZE-2
NCT06066528
3 Overweight / obesity with type 2 diabetes 755 3.6 and 6.0 mg SC Once weekly 76 weeks Phase 3 randomized evaluation of survodutide versus placebo in participants with overweight or obesity and type 2 diabetes. ClinicalTrials.gov
Cirrhosis PK / Safety Study
NCT05296733
2 Participants with compensated or decompensated cirrhosis and overweight / obesity cohorts 41 in multiple-dose cohorts Escalated study doses up to 6.0 mg SC Once weekly 28 weeks Study evaluated pharmacokinetics and safety and reported reductions in several liver-related and metabolic measures. PubMed

Doses Studied in Survodutide Clinical Research

Survodutide has been studied using different dose-escalation schemes depending on the indication and trial. Published phase 2 obesity research evaluated 0.6–4.8 mg once weekly, while later phase 3 obesity studies evaluated 3.6 mg and 6.0 mg once-weekly treatment groups.

Research context Doses documented Status
Type 2 diabetes — Phase 2 Up to 0.3, 0.9, 1.8 or 2.7 mg once weekly; additional 1.2 and 1.8 mg twice-weekly groups were evaluated. TRIAL DOSES
Obesity — Phase 2 0.6, 2.4, 3.6 and 4.8 mg once weekly. TRIAL DOSES
MASH — Phase 2 2.4, 4.8 and 6.0 mg once weekly. TRIAL DOSES
SYNCHRONIZE-1 — Phase 3 3.6 and 6.0 mg once weekly. PHASE 3
SYNCHRONIZE-2 — Phase 3 3.6 and 6.0 mg once weekly. PHASE 3
FDA-approved dosage No FDA-approved survodutide dosage exists. NOT APPROVED

Trial doses are reported to describe the clinical literature. They are not individualized dosing recommendations.

Survodutide Research Outcomes

Body Weight

In the 46-week phase 2 obesity study, mean body-weight reductions increased across the studied survodutide groups. The planned-treatment analysis reported −6.2%, −12.5%, −13.2% and −14.9% changes with 0.6, 2.4, 3.6 and 4.8 mg, respectively, compared with −2.8% with placebo.

Phase 3 Obesity Evidence

The 2026 SYNCHRONIZE-1 publication reported mean body-weight changes at week 76 of −12.2% with 3.6 mg and −13.0% with 6.0 mg survodutide, compared with −5.4% with placebo.

Glycemic Control

In a randomized phase 2 type 2 diabetes study, survodutide produced reductions in HbA1c across several studied dose groups. Body-weight reduction also showed a dose-related pattern, with the highest twice-weekly group producing an approximately 8.7% mean reduction in body weight at week 16.

MASH and Liver Outcomes

A 48-week phase 2 MASH trial randomized 293 participants with biopsy-confirmed MASH and fibrosis stages F1 through F3. Histologic improvement in MASH without worsening of fibrosis occurred in 47%, 62% and 43% of participants receiving 2.4, 4.8 and 6.0 mg, respectively, compared with 14% with placebo.

Dose-Response Relationships

Dose-response patterns have differed according to endpoint and indication. The phase 2 obesity trial showed progressively greater mean weight reduction across several dose groups, while the MASH study's primary histologic endpoint was modeled using a nonlinear dose-response relationship.

Interpretation: Clinical dose-response findings describe populations and protocols studied under controlled conditions. They do not establish a universally optimal dose for an individual.

Adverse Events & Safety Findings

Gastrointestinal adverse events are prominent across published survodutide trials. In the phase 2 obesity study, gastrointestinal adverse events occurred in approximately 75% of survodutide-treated participants compared with 42% of placebo recipients.

In the 2026 SYNCHRONIZE-1 phase 3 study, gastrointestinal symptoms occurred in 80.9% of participants receiving 3.6 mg and 89.7% of participants receiving 6.0 mg, compared with 47.9% receiving placebo. The published report characterized these events as typically mild to moderate.

In the phase 2 MASH study, nausea occurred in 66% of participants receiving 2.4 mg, diarrhea in 49%, and vomiting in 41%; corresponding placebo rates were 23%, 23% and 4%.

Investigational safety status: Because survodutide is still investigational, the clinical safety profile continues to be characterized through ongoing and completed trials. There is no FDA-approved product label that should be treated as the definitive clinical-use instruction for survodutide.

Research Limitations & What Remains Unknown

Survodutide remains under clinical development. Although phase 2 studies and emerging phase 3 results provide substantial evidence, trial results remain population- and protocol-specific.

Important unanswered questions include long-term safety, comparative effectiveness against established metabolic therapies, optimal treatment strategies, durability of treatment effects, long-term liver outcomes and the eventual regulatory status of individual indications.

MASH and liver-related results are particularly important to interpret carefully because histologic and biomarker endpoints do not by themselves establish long-term clinical outcomes.

The transition from phase 2 evidence to phase 3 evidence also does not guarantee regulatory approval. Regulatory decisions depend on the totality of efficacy, safety, manufacturing and other evidence submitted to the applicable authority.

BAC Water / Reconstitution Considerations

There is no universal BAC-water volume for “survodutide” as a compound.

Survodutide is investigational, and published clinical trials should not be interpreted as establishing a universal preparation method for unrelated research vials.

Formulation-specific information matters. If a particular investigational study or supplied research material specifies a formulation, concentration, vehicle, preparation procedure or stability condition, that information belongs to that specific formulation and should not automatically be generalized to another vial.

General Concentration Formula

Total vial quantity ÷ final liquid volume = concentration

For example, 10 mg divided by 2 mL equals 5 mg/mL mathematically. This is a concentration calculation only and does not establish an appropriate preparation, administration volume or human dose.

Quantity Final volume Mathematical concentration
5 mg 1 mL 5 mg/mL
10 mg 2 mL 5 mg/mL
15 mg 1.5 mL 10 mg/mL
20 mg 2 mL 10 mg/mL
30 mg 3 mL 10 mg/mL

Survodutide Concentration Calculator

Use the BacScience universal calculator for mathematical concentration calculations.

Calculate Concentration

Continue Your Research

Survodutide Research FAQ

Is survodutide FDA approved?

No. Survodutide (BI 456906) is an investigational compound in clinical development. It does not have an FDA-approved indication or FDA-approved dosage.

What is survodutide?

Survodutide is an investigational dual agonist of the glucagon receptor and GLP-1 receptor being studied in obesity, type 2 diabetes, MASH and related metabolic conditions.

What receptors does survodutide activate?

Survodutide activates both the glucagon receptor and the glucagon-like peptide-1 receptor.

What survodutide doses have been studied?

Clinical studies have evaluated different regimens. The phase 2 obesity study evaluated 0.6, 2.4, 3.6 and 4.8 mg once weekly, while later phase 3 obesity studies evaluated 3.6 and 6.0 mg once weekly.

What did the phase 2 obesity study find?

At week 46, mean body-weight changes were approximately −6.2%, −12.5%, −13.2% and −14.9% with 0.6, 2.4, 3.6 and 4.8 mg, respectively, compared with −2.8% with placebo.

What did the phase 3 SYNCHRONIZE-1 study find?

At week 76, mean body-weight change was approximately −12.2% with 3.6 mg and −13.0% with 6.0 mg survodutide compared with −5.4% with placebo in adults with obesity without diabetes.

Has survodutide been studied for MASH?

Yes. A randomized phase 2 trial enrolled 293 participants with biopsy-confirmed MASH and fibrosis stages F1 through F3 and evaluated 2.4, 4.8 and 6.0 mg once weekly for 48 weeks.

How much BAC water should be used for survodutide?

There is no universal BAC-water volume that applies to every survodutide research material. Concentration mathematics depends on the total quantity and final liquid volume, while an actual preparation method depends on the specific formulation and experimental requirements.

How do you calculate survodutide concentration?

Divide total compound quantity by final liquid volume. For example, 10 mg divided by 2 mL equals 5 mg/mL mathematically. This is a concentration calculation, not a dosing recommendation.

Is a clinical-trial dose a recommended dose?

No. A clinical-trial dose describes the protocol used in a particular study population. It should not be converted into a personalized treatment recommendation.

What are the most common adverse events reported with survodutide?

Gastrointestinal adverse events, including nausea, diarrhea and vomiting, are prominent in published clinical trials. Frequency varies according to dose, study population and treatment period.

Where can researchers verify current survodutide clinical status?

Current clinical status should be checked using ClinicalTrials.gov, PubMed and the primary journal publications. Regulatory status should be verified through the applicable regulatory authority.

Scientific References

  1. le Roux CW, et al. Glucagon and GLP-1 receptor dual agonist survodutide for obesity: a randomised, double-blind, placebo-controlled, dose-finding phase 2 trial. Lancet Diabetes & Endocrinology. 2024. PubMed .
  2. Blüher M, et al. Dose-response effects on HbA1c and bodyweight reduction of survodutide, a dual glucagon/GLP-1 receptor agonist, compared with placebo and open-label semaglutide in people with type 2 diabetes. Diabetologia. 2024. PubMed .
  3. Sanyal AJ, et al. A Phase 2 Randomized Trial of Survodutide in MASH and Fibrosis. New England Journal of Medicine. 2024. PubMed .
  4. le Roux CW, et al. Survodutide Once Weekly for the Treatment of Adults with Obesity. New England Journal of Medicine. 2026. PubMed .
  5. ClinicalTrials.gov. SYNCHRONIZE-1: NCT06066515. ClinicalTrials.gov .
  6. ClinicalTrials.gov. SYNCHRONIZE-2: NCT06066528. ClinicalTrials.gov .
  7. Ekinci EI, et al. Dual Glucagon and GLP-1 Receptor Agonist Survodutide Improves Biomarkers of Beta-Cell Function and Insulin Sensitivity. Diabetes, Obesity and Metabolism. 2026. PubMed .
  8. Survodutide pharmacokinetic and safety research in cirrhosis. Journal of Hepatology / related clinical publication. PubMed .

Last scientifically reviewed: September 11, 2026. This page is an educational clinical-research reference and is not medical advice, a prescribing guide, or a human treatment protocol.